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oralanabolic

Furazabol

MiotolanTPA17α-methyl-5α-androstano[2,3-c]furazan-17β-ol
270
Anabolic Ratio
73
Androgenic Ratio
0.33d
Half-Life

Unique Japanese compound synthesized in the 1960s with a furazan (1,2,5-oxadiazole) ring fused to the A-ring. Historically used for hyperlipidemia as well as anemia — one of the few AAS that actually lowers LDL cholesterol rather than raising it. Used by several Eastern Bloc athletes in the 1980s-90s.

Mechanism of Action

DHT-derived with the 2,3-furazan ring modification. Does not aromatize. 17α-methylated for oral bioavailability. The furazan ring uniquely alters lipid metabolism — documented LDL reduction mechanism via hepatic lipase modulation, distinct from typical AAS lipid effects. Moderate AR agonism with reasonable anabolic:androgenic selectivity.

Furazabol molecule
Molecular structure

Typical Dosing

56 mg
low / week
112 mg
moderate / week
210 mg
high / week

⚠ Warning Flags

  • Very rare outside Japan — significant counterfeiting risk
  • Limited modern clinical data

Effect Profile

Muscle Protein Synthesis
4Low
Nitrogen Retention
4Low
Strength Gains
5Moderate
Red Blood Cell Production
3Low
Fat Loss
4Low
Glycogen Storage
4Low
Recovery Speed
4Low
Collagen Synthesis
3Low

Side Effect Profile

Hormonal Suppression
5Moderate
Estrogenic Effects
1Minimal
Androgenic Effects
3Low
Cardiovascular Strain
3Low
Liver Stress
4Low
Insulin Resistance
2Minimal
Mood Changes
3Low
Prostate Risk
2Minimal

Research Studies

Anabolic and androgenic effects of furazabol and its effect on serum lipids

Yamamoto I, et al. · 1969

PubMed

Original pharmacological characterization of furazabol showing anabolic:androgenic ratio of ~3.7:1 (Hershberger) with the unique finding of LDL cholesterol reduction — contrasting with other AAS of the era that universally worsened lipid profiles.