Compound Database

The gold-standard reference anabolic compound. Long-ester testosterone providing stable blood levels with weekly or bi-weekly injections. Used as the base of virtually all cycles.

Nearly identical to Testosterone Enanthate in effects and side effects. Slightly longer half-life (8 days). The most commonly prescribed TRT form in the USA.

Short-ester testosterone requiring every-other-day (EOD) or even daily injections. Faster onset, shorter clearance time — popular for shorter cycles or pre-contest use.

One of the most studied synthetic AAS. Highly anabolic, low androgenic. Excellent for joint health and lean mass. Very suppressive. Famous 'Deca Dick' risk from prolactin/progestin activity.

The short-ester version of nandrolone — the original nandrolone formulation (1959) before Deca's longer decanoate ester became dominant. Identical pharmacology to Deca-Durabolin but with a 2.5-day half-life requiring every-other-day (EOD) or twice-weekly injections. Preferred by experienced users who want faster blood level control, quicker cycle clearance, and the ability to adjust dose rapidly. Side effects clear faster if issues arise.

One of the most clinically used anabolic steroids in the world. Extremely mild androgenic profile. Used medically for burn recovery, HIV wasting, pediatric growth. Minimal liver stress for an oral. Expensive.

DHT-derived oral. Dry, hard look — no water retention. Poor joint lubrication (notorious for joint pain). Significant cholesterol impact. Often used pre-contest for vascularity.

The original mass-builder. Extremely fast strength and size gains, significant water retention. The first synthetic AAS mass-produced for athletic performance. Liver toxic, highly estrogenic.

The most potent anabolic steroid available. No aromatization but strong progestogenic activity. Extreme muscle hardness and fat loss. Notorious for severe side effects: 'Tren cough', night sweats, insomnia, aggression, cardio impairment.

Veterinary testosterone derivative. Slow-acting, lean mass with improved vascularity. Significantly increases appetite and red blood cell production (EPO-like effect). Low estrogenic activity.

One of the mildest and most tolerable anabolic steroids. No aromatization, very low androgenic effect. Mild suppression. Ideal for women or those sensitive to side effects. Weak mass builder alone — best combined.

DHT-derivative with anti-estrogenic properties (weak aromatase inhibitor). Produces muscle density and hardness with no water retention. Best at low body fat. Often used as a 'dry' compound finishing cycle.

East German Olympic program compound. Modified Dianabol with a chloro group that prevents aromatization. Steady, lean gains without water retention. Less liver stress than Dianabol but still oral 17α-alkylated.

One of the most potent oral AAS ever developed. Originally prescribed for anemia and muscle-wasting diseases. Produces the fastest and most dramatic strength and mass gains of any oral compound. Severely hepatotoxic and strongly suppressive.

DHT-derived oral androgen with uniquely low anabolic muscle tissue activity due to extensive inactivation by 3α-HSD in muscle. Used clinically for male hypogonadism and infertility. Valued as a cycle ancillary: lowers SHBG (freeing more testosterone), mildly inhibits aromatase, and improves mood/libido.

One of the most potent androgens ever synthesized. Extremely high androgenic:anabolic ratio. Used medically for hypogonadism, delayed puberty, and breast cancer. In sports: pure aggression and strength compound with negligible mass gains. Carries severe health risks.

Experimental compound originally developed as a male contraceptive by the Population Council. Extremely potent anabolic — roughly 10× more myotrophic than testosterone. Aromatizes at a higher rate than testosterone. Does not reduce to DHT (no 5α-reduction). Potent HPTA suppression — used as contraceptive precisely because it shuts down sperm production completely.

2α,17α-dimethyl-5α-androstan-17β-ol-3-one. Originally synthesized in the 1950s by Syntex but never marketed. Re-emerged in 2005 as an 'over-the-counter' designer steroid. Extremely potent oral — mg-for-mg one of the strongest available. No aromatization, no estrogenic activity. Very hepatotoxic despite mild androgenic profile.

17α-methylated version of 1-testosterone (1-dehydrotestosterone). Regarded as one of the most potent oral AAS per milligram. Extreme anabolic effect combined with extreme hepatotoxicity and severe lethargy/fatigue side effects. Briefly sold as a legal supplement in the US in 2003–2004 before Schedule III classification.

Designer prohormone/AAS briefly sold legally in the US (2010–2012) before DEA scheduling. 2-methyl, 17α-methyl DHT derivative. Produces rapid strength and size gains. Very hepatotoxic with notable cardiovascular impact. Limited human pharmacokinetic data available.

Designer AAS developed by Patrick Arnold (BALCO scandal). First identified in an athlete's sample in 2003, leading to one of the most significant doping scandals in sports history. Not a natural steroid derivative — purely synthetic. Limited clinical data due to its illicit nature.

One of the first synthetic anabolic steroids developed (1956), predating Dianabol. A 19-nor compound (related to nandrolone) with an ethyl group at C17. Historically used for anemia and muscle wasting. Notable for being the first synthetic progestogen to show potent anabolic properties. Now largely replaced by safer alternatives.

4-chloro modified testosterone. Unable to aromatize (4-chloro blocks aromatase) and resists 5α-reduction. Very mild anabolic steroid used medically in Italy and Brazil as a topical preparation. Caused a major doping scandal in 2024 when tennis player Jannik Sinner tested positive via contaminated spray.

Unique Japanese compound synthesized in the 1960s with a furazan (1,2,5-oxadiazole) ring fused to the A-ring. Historically used for hyperlipidemia as well as anemia — one of the few AAS that actually lowers LDL cholesterol rather than raising it. Used by several Eastern Bloc athletes in the 1980s-90s.

17α-methylated dihydrotestosterone. Purely androgenic with only modest anabolic activity in muscle (like all DHT derivatives, it is inactivated by 3α-HSD in muscle). Historically used for hypogonadism in Germany (Ermalone). Produces pronounced strength and aggression effects with limited mass gains.

2-methyl-5α-dihydrotestosterone derivative (injectable). Mildly anabolic with low androgenic activity and no aromatization. Produces lean, dry gains similar to Primobolan. Historically marketed in Spain (Anatrofin) and Italy. Now extremely rare but still discussed in harm-reduction circles for its clean side-effect profile.

17α-methyl-5-androstene-3β,17β-diol — a testosterone and DHEA derivative used medically in the 1960s–1970s. Aromatizes to methylestradiol. Notable for a reported synergistic effect when combined with other AAS, though this claim lacks rigorous modern documentation. Rarely used today.

Novel investigational compound under active clinical development (NIH/Los Angeles Biomedical Research Institute) as a once-daily oral male contraceptive. Uniquely combines androgenic and progestogenic activity — meaning a single agent can fully suppress spermatogenesis without requiring a progestin co-agent. Not available outside clinical trials.

First identified in a competitive cyclist's urine in 2002 by WADA — the first 'designer steroid' discovered in the modern era of doping. Originally synthesized by Wyeth in the 1960s but never marketed. Patrick Arnold (BALCO) reportedly distributed it before THG gained notoriety. Exceptionally high anabolic:androgenic ratio.

4-chlorinated 19-nortestosterone derivative combining the aromatization blockade of clostebol with the reduced androgenicity of 19-nor compounds. Very mild compound with extremely low androgenic activity. Used medically in France and some European countries for anemia. Very limited modern data.

4-hydroxy-19-nortestosterone cipionate — an injectable 19-nor AAS with a hydroxyl group at C4. Very low androgenic activity. Marketed in Italy (Sterobol) for anemia and muscle-wasting conditions. Essentially non-virilizing at therapeutic doses, making it one of the most androgenically mild compounds available.

Prohormone of 19-nortestosterone (nandrolone) — converts in vivo to nandrolone via enzymatic oxidation. Marketed briefly as an OTC prohormone supplement in the late 1990s/2000s (as 19-norandrostenediol) following the DSHEA loophole era. Effects are those of nandrolone but attenuated by incomplete conversion.

The most abundant steroid hormone in the human body. A naturally occurring adrenal precursor that converts to testosterone and estradiol in peripheral tissues. Sold OTC in the US as a dietary supplement. Modest effects at physiological doses — used primarily by older adults for age-related testosterone decline. Prohibited by WADA in sport.

Naturally occurring androgen/estrogen precursor produced by the adrenal glands, ovaries, and testes. Made famous by Mark McGwire's 1998 home-run season when he admitted using it as a supplement. Peak serum testosterone elevation is modest and transient (~3 hours). Banned by MLB in 2004 and classified Schedule III in the US in 2004.

Natural prohormone that converts to testosterone via 3β-HSD. Produced naturally in the adrenal cortex and gonads. Sold briefly as an OTC supplement in the post-DSHEA era (late 1990s). More immune-stimulating effects documented than true anabolic effects. Minimal ergogenic benefit established in human trials. Scheduled III in 2004.

Recombinant human growth hormone. A 191-amino-acid peptide hormone that stimulates IGF-1 production in the liver, driving fat loss, collagen synthesis, recovery, and modest lean mass gains via indirect anabolic pathways.

Exogenous insulin used in bodybuilding to shuttle glucose and amino acids into muscle cells. Extremely potent at glycogen supercompensation and anti-catabolism but carries the highest acute lethality risk of any PED due to hypoglycemia.

A modified version of insulin-like growth factor 1 with extended half-life. Mediates most of GH's anabolic effects, directly stimulating muscle cell growth via PI3K/Akt/mTOR signaling with theoretical hyperplasia potential.

A synthetic GHRH (growth hormone-releasing hormone) analog that stimulates the pituitary to release endogenous growth hormone. The DAC variant has an extended half-life of ~8 days, creating sustained GH elevation.

A selective growth hormone secretagogue (ghrelin receptor agonist) that stimulates pituitary GH release without significantly elevating cortisol or prolactin — considered the cleanest GH peptide available.

A potent ghrelin mimetic that strongly stimulates GH release and appetite. Unlike Ipamorelin, GHRP-6 also elevates cortisol and prolactin and causes extreme hunger, making it best suited for bulking phases.

A non-aromatizing injectable anabolic steroid (DHT derivative of Boldenone). Provides lean, dry gains similar to Primobolan but with stronger anabolic potency. Known for severe post-injection pain (PIP). Very limited human clinical data exists.

Pure testosterone in aqueous suspension with no ester attached. Produces immediate, very high peak testosterone levels within hours of injection. Used primarily pre-workout or pre-competition for rapid strength and aggression. Requires daily (or more frequent) injections.

Long-ester version of Trenbolone. Same parent hormone as Trenbolone Acetate but with a longer half-life requiring less frequent injections. Extremely potent 19-Nor with strong AR binding, anti-catabolic glucocorticoid receptor antagonism, and local IGF-1 elevation. Reserved for experienced users only.

Long-ester version of Drostanolone (Masteron). A DHT derivative with anti-estrogenic properties, commonly used during contest prep for a hard, dry cosmetic look. Longer half-life than Masteron Propionate allows less frequent injections.

An oral testosterone formulation that bypasses first-pass liver metabolism via lymphatic absorption. The only orally bioavailable testosterone that is not 17α-alkylated, making it significantly less hepatotoxic than other oral steroids. Also available as a very long-acting injectable (Nebido).

An extremely potent oral androgen originally developed as a veterinary drug to suppress estrus in dogs. Used by some powerlifters and fighters pre-competition for its powerful CNS-stimulating aggression effects. One of the most hepatotoxic steroids in existence — extremely dangerous with very limited practical application.