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oralanabolic

Methyl-1-Testosterone

M1T17α-methyl-1-testosterone
910
Anabolic Ratio
100
Androgenic Ratio
0.17d
Half-Life

17α-methylated version of 1-testosterone (1-dehydrotestosterone). Regarded as one of the most potent oral AAS per milligram. Extreme anabolic effect combined with extreme hepatotoxicity and severe lethargy/fatigue side effects. Briefly sold as a legal supplement in the US in 2003–2004 before Schedule III classification.

Mechanism of Action

5α-reduced, 17α-methylated testosterone derivative — already in its most potent AR-binding form (cannot be 5α-reduced further). Does not aromatize. AR binding affinity far exceeding testosterone. The severe lethargy is mechanistically unexplained but is reported universally. Hepatotoxic through the standard 17α-alkyl first-pass metabolism pathway.

Methyl-1-Testosterone molecule
Molecular structure

Typical Dosing

35 mg
low / week
70 mg
moderate / week
140 mg
high / week

⚠ Warning Flags

  • Extreme hepatotoxicity at doses as low as 5 mg/day
  • Severe lethargy/fatigue reported by virtually all users
  • Extremely suppressive — extended PCT required
  • Schedule III controlled substance in the US (2004)

Effect Profile

Muscle Protein Synthesis
8Very High
Nitrogen Retention
8Very High
Strength Gains
8Very High
Red Blood Cell Production
3Low
Fat Loss
4Low
Glycogen Storage
6Moderate
Recovery Speed
7High
Collagen Synthesis
4Low

Side Effect Profile

Hormonal Suppression
8Very High
Estrogenic Effects
2Minimal
Androgenic Effects
5Moderate
Cardiovascular Strain
6Moderate
Liver Stress
8Very High
Insulin Resistance
5Moderate
Mood Changes
7High
Prostate Risk
4Low

Research Studies

Comparative anabolic potency of testosterone, 1-testosterone, and M1T in rodent models

Jasuja GK, et al. · 2005

PubMed

M1T showed 9× greater levator ani muscle weight gain than testosterone at equivalent doses in rodent models, corroborating its extreme anabolic:androgenic ratio and establishing the pharmacological basis for its potency.