← Back to Compounds
injectablepeptide

Ipamorelin

IpamIPAMNNC 26-0161
0
Anabolic Ratio
0
Androgenic Ratio
0.08d
Half-Life

A selective growth hormone secretagogue (ghrelin receptor agonist) that stimulates pituitary GH release without significantly elevating cortisol or prolactin — considered the cleanest GH peptide available.

Mechanism of Action

Acts as a selective agonist of the ghrelin receptor (GHS-R1a) on pituitary somatotroph cells. Stimulates endogenous GH release while preserving normal pulsatile GH secretion patterns. Uniquely selective — does not significantly stimulate ACTH, cortisol, or prolactin release unlike other GHRP peptides. Downstream effects mediated through the GH/IGF-1 axis.

Ipamorelin molecule
Molecular structure

Typical Dosing

1.4 mg
low / week
2.1 mg
moderate / week
4.2 mg
high / week

⚠ Warning Flags

  • Mild water retention possible
  • Mild appetite increase
  • Best used in combination with CJC-1295 for synergistic GH release

Effect Profile

Muscle Protein Synthesis
3Low
Nitrogen Retention
3Low
Strength Gains
2Minimal
Red Blood Cell Production
1Minimal
Fat Loss
5Moderate
Glycogen Storage
3Low
Recovery Speed
6Moderate
Collagen Synthesis
5Moderate

Side Effect Profile

Hormonal Suppression
1Minimal
Estrogenic Effects
1Minimal
Androgenic Effects
1Minimal
Cardiovascular Strain
1Minimal
Liver Stress
1Minimal
Insulin Resistance
2Minimal
Mood Changes
1Minimal
Prostate Risk
1Minimal

Research Studies

Ipamorelin, the first selective growth hormone secretagogue

Raun K et al. · 1998

PubMed

Demonstrated that Ipamorelin selectively stimulates GH release without increasing cortisol or prolactin — unique among GH secretagogues.

Pharmacokinetic-pharmacodynamic modeling of ipamorelin, a growth hormone releasing peptide, in human volunteers

Gobburu JV et al. · 1999

PubMed

Characterized the dose-response pharmacokinetics of Ipamorelin GH secretion in healthy humans.

Growth hormone-releasing peptide (GHRP)

Bowers CY · 2012

PubMed

Comprehensive review of growth hormone secretagogues and their clinical effects on the somatotropic axis.